TMEM97 regulates cholesterol biosynthesis and mitochondrial metabolism in gastric carcinoma

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Authors: Saniye Koç Ada, Yahya Yozbatıran, Bengü Yaren Beyaz, Ceren Sarı, Elif Gelenli Dolanbay, Halil İbrahim Saygı, Ceren Sümer, Cyrille Mesue Njume, Ali Çakmak, Fatma Zehra Sarı, Şeyma Çimen, Mertkaya Aras
+2 more Andaç Kılıçkap, Burcu YUCEL
Cited by: 1
References: 47

Abstract

Cancer cells regulate cholesterol levels to sustain proliferation and metabolic adaptation. Here, we show that TMEM97 coordinates cholesterol sensing with oxidative phosphorylation in gastric cancer. Loss of TMEM97 reduced in vitro proliferation and impaired xenograft tumor growth. TMEM97 deficiency disrupted cholesterol homeostasis, causing accumulation of the post-lanosterol intermediate follicular fluid meiosis-activating sterol (FF-MAS) under both normal and lipoprotein-deficient serum conditions. Metabolomic and transcriptomic analyses further revealed altered tricarboxylic acid (TCA) cycle activity under lipoprotein-deficient conditions. Structural analysis identified a conserved cholesterol recognition amino acid consensus (CARC) motif in TMEM97, and its deletion reduced cholesterol binding. Collectively, TMEM97 regulates sterol regulatory element binding protein (SREBP)-associated lipid programs, post-lanosterol cholesterol biosynthesis, and mitochondrial bioenergetics. These findings identify TMEM97 as a regulator of cholesterol-dependent metabolic adaptation in gastric cancer and support TMEM97-associated cholesterol regulation as a potential therapeutic vulnerability.

Fields of Study & Subjects

Cholesterol and Lipid Metabolism Ferroptosis and cancer prognosis Caveolin-1 and cellular processes

Publication details

Journal / source
iScience
Publisher
Elsevier BV
Volume
29
Issue
8
Pages
116773–116773
Publication date
14 July 2026
Language
EN