Presynaptic Terminal Proteins and Nicotinic Receptors Are Depleted From Mouse Parasympathetic Ganglionic Junctions Paralyzed With Botulinum Neurotoxin Type A

Toxicon 2026

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Authors: Ahmed Al-Sabi, Gary W. Lawrence

DOI: 10.1016/j.toxicon.2025.108797

Cited by: 1

Fields of Study & Subjects

Botulinum Toxin and Related Neurological Disorders Nicotinic Acetylcholine Receptors Study Pain Mechanisms and Treatments