A high-throughput, quantitative platform using 2D dissociated human cerebral organoids to model neuroinflammation in Alzheimer’s disease

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Authors: Meagan N. Olson, Nathaniel Barton, Luyao Feng, Samantha M. Chigas, Khanh V. Tran, Adrian R. Orszulak, Jafira M. Johnson, Pepper Dawes, Chandani Shrestha, Vishali R. Umaiyalan, Yen-Hsiang Huang, Jonathan Sundstrom
+8 more Liam F. Murray, Q. Wang, Hyung Suk Oh, Megan H. Orzalli, David M. Knipe, Benjamin Readhead, Yingleong Chan, Elaine T. Lim
Cited by: 3
References: 141

Abstract

Neuroinflammation is a key process associated with Alzheimer's disease (AD). There is interest in developing New Approach Methodologies (NAMs) by using human in-vitro complex systems such as brain organoids, combined with machine learning and computational approaches, to reproducibly and robustly evaluate monoclonal antibodies and other therapeutic modalities on these human-derived systems. Herpesviruses such as herpes simplex virus 1 (HSV-1) had been shown to be associated with AD risk and molecular pathology. Building on top of previously reported work, we used herpes simplex virus 1 (HSV-1) infection in 2D dissociated cells from human cerebral organoids (dcOrgs) to recapitulate AD-associated molecular readouts, such as high co-abundance of intracellular beta amyloid (Aβ) and phosphorylated tau (pTau) with HSV-1. Secreted Aβ42/40 ratios in conditioned media were lower from HSV-1-infected dcOrgs, compared to mock dcOrgs. Differentially expressed transcripts from bulk and single-cell RNA sequence data in HSV-1-infected dcOrgs were enriched for AD-associated GWAS genes. Our high-throughput, quantitative framework represents a comprehensive approach to harness on the strengths of 2D dcOrgs for high-throughput applications such as therapeutic screens and can complement the 3D brain organoids and animal models for neuroinflammation in AD.

Fields of Study & Subjects

Single-cell and spatial transcriptomics Alzheimer's disease research and treatments Neuroinflammation and Neurodegeneration Mechanisms

Publication details

Journal / source
npj Dementia
Publisher
Springer Science and Business Media LLC
Volume
2
Issue
1
Pages
20–20
Publication date
9 March 2026
Language
EN