Correction: Loss of the tumour suppressor LKB1/STK11 uncovers a leptin-mediated sensitivity mechanism to mitochondrial uncouplers for targeted cancer therapy

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Authors: Andriani Angelopoulou, Giorgos Theocharous, Dimitrios Valakos, Aikaterini Polyzou, Sophia Magkouta, Vassilios Myrianthopoulos, Sophia Havaki, Marco Fiorillo, Ioanna Tremi, Κonstantinos Vachlas, Theodoros Nisotakis, Dimitris-Foivos Thanos
+8 more Αnastasia A. Pantazaki, Dimitris Kletsas, Jiri Jr Bartek, Petty Rd, Dimitris Thanos, Rory J. McCrimmon, Angelos Papaspyropoulos, Vassilis G. Gorgoulis
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References: 1

Abstract

Correction: Mol Cancer 23, 147 (2024) Following publication of the original correspondence [1], the authors wish to extend their acknowledgements to provide a more clear and precise credit to Dr P Haramis, Dr L Mans and Dr G Gargiulo’s work that was included in their manuscript. First, they would like to thank Dr P Haramis and Dr L Mans for the work done in zebrafish and cite the data published in Dr Mans’ doctoral thesis [2] used in Figs. 1 C-F, S1B-E and S2 of the manuscript. They also thank Dr Haramis for her original concept of exploiting the hypermetabolic phenotype of lkb1 mutant zebrafish in a synthetic lethality screen. Finally, they would like to extend their acknowledgements to Dr G Gargiulo for the analysis of the RNA-seq experiments on zebrafish performed at the Genomics Core Facility of the NKI.

Fields of Study & Subjects

Metabolism Diabetes and Cancer Cancer Hypoxia and Metabolism Lipids

Publication details

Journal / source
Molecular Cancer
Volume
25
Issue
1
Pages
11–11
Publication date
12 January 2026
Language
EN