Amyloid Beta Revisited: A Versatile Immune Polypeptide
Abstract
Alzheimer's disease affects about fifty million people worldwide. Its main symptoms are the formation of amyloid deposits and of neurofibrillary tangles, and cognition loss. To date, no clear etiology for this disease could be identified. I will here elaborate on the likelihood that Amyloid b is not a toxic molecule but a protective protein fragment, and that a combination of chemical and biological exposures, with metals playing a key role, are the triggering factors for Alzheimer's type dementia.
Fields of Study & Subjects
Alzheimer's disease research and treatments
Dementia and Cognitive Impairment Research
Neuroinflammation and Neurodegeneration Mechanisms
Publication details
- Journal / source
- Qeios
- Publication date
- 7 March 2026
- Language
- EN
- Document type
- Preprint