Prediction, validation, and learning for mitochondrial-damaged tumor cells

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Authors: Yanagida Shozo, Murakami Noriyuki, Nakagawa Juro
Cited by: 0

Abstract

Abstract Through Molecular Modeling using software Spartan, we learned about cell engine. i.e., mitochondria. the following: (1) Chemical energy hydrated superoxide anion radical [O2-*(H2O)2] is generated in mitochondrial endoplasmic reticulum lipid bilayer, (2) This chemical energy [[O2-*(H2O)2]] generates a capacitive current in the mitochondrial membrane. (3) Capacitive current between mitochondrial membranes generates microwave and radio frequency energy. (4) Microwave and radio frequency energy which are identical with infrared (IR) and far infrared (FIR) energy, i.e., thermal energy, which promotes thermal metabolic reactions in cells. (5) When the chemical energy [O2-*(H2O)2]is overproduced, and undergoes intra molecular disproportionation reaction, hydrated hydroxyl radical [HO*(H2O) 2], the main cause of cell aging,(Cellular Theory) is generated in the bilayer mitochondrial membranes, causing major changes in the molecular membrane structure, leading to cellular enlargement. It is also verified that iodine therapy for cancer is due to the excellent antioxidant effect of iodide ion. Assumed the cause of cancer is cellular aging, cells with active mitochondrial reticulum activated by iodide ion are immune cells, and cells with enlarged, and degraded mitochondrial reticulum are tumor cells.

Fields of Study & Subjects

Cancer and biochemical research Mitochondrial Function and Pathology Redox biology and oxidative stress

Publication details

Journal / source
Zenodo (CERN European Organization for Nuclear Research)
Publication date
17 January 2026
Language
EN