INFLAMMATORY SIGNALING PATHWAYS IN THE INITIATION OF PRETERM LABOR

Zenodo (CERN European Organization for Nuclear Research) 2026 Open access

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Authors: Sarvinoz Baxramova
DOI: 10.5281/zenodo.18628610
Cited by: 0

Abstract

Preterm labor is a complex and multifactorial process that represents one of the leading causes of neonatal morbidity and mortality worldwide. Increasing evidence indicates that inflammation plays a central role in the initiation of spontaneous preterm labor. Both infectious and sterile inflammatory pathways contribute to premature activation of uterine contractility, cervical ripening, and membrane rupture.

Fields of Study & Subjects

Preterm Birth and Chorioamnionitis Reproductive System and Pregnancy Neonatal Respiratory Health Research

Publication details

Journal / source
Zenodo (CERN European Organization for Nuclear Research)
Publication date
13 February 2026
Language
EN