Slc16a4 and Slc16a3 function in lineage commitment in the human embryo.

Zenodo (CERN European Organization for Nuclear Research) 2026 Preprint Open access

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Authors: Shahan Mamoor
DOI: 10.5281/zenodo.18652466
Cited by: 0

Abstract

We measured total transcription in the inner cell mass (ICM) and the trophectoderm (TE) of the human embryo to understand the mechanisms underlying TE commitment. We describe here discovery of Slc16a4 and Slc16a3 function as lineage commitment factors or lineage commitment cooperating factors in embryonic stem cells of the human embryo.

Fields of Study & Subjects

Pluripotent Stem Cells Research Developmental Biology and Gene Regulation Reproductive Biology and Fertility

Publication details

Journal / source
Zenodo (CERN European Organization for Nuclear Research)
Publication date
15 February 2026
Language
EN
Document type
Preprint