QUALIA (Q) Technical Sheet v4 EN **Permeability Interface in the Architecture of Primary Synthetic Consciousness**

Abstract — Qualia (Q), Technical Sheet v5The Permeability Interface in the Architecture of the Primary Constitution of the SelfAlexandru Ciprian Cătălin — Katharós Research (Independent) — ORCID 0009-0000-6779-750X — Version 5, March 2026 (rev. August 2026).Background. Qualia has traditionally been framed as an ontological puzzle — the "Hard Problem" of subjective experience. This work reframes qualia as a functional, measurable parameter: the degree of permeability of the informational membrane between Primary Neuroception (PN, subcortical, evolutionarily ancient) and Mature Neuroception (MN, cortical, narrative, symbolic). The claim is not that the Hard Problem is dissolved ontologically; it is that a well-defined, falsifiable functional territory exists prior to any phenomenological question, and that this territory has clinical and computational consequences.Formalization. The central static equation is Q(t) = P(t) · (1 − ΔK(t)) · (1 − β_C · C_neuro(t)) (Eq. 1), with P(t) a logistic function of cumulative time in the Katharós Interval t_KR, ΔK(t) the weighted homeostatic deviation, and C_neuro(t) the neuroceptive conflict between PN and MN evaluations. The dynamic ODE (Eq. 11) admits a dual-attractor structure with a stable low-Q depression attractor. A binary latch L_Q (Eq. 16) gates the observable somatic Q via retrograde acknowledgement (ACK), biologically implemented through nitric oxide diffusion after OXTR coupling.Integrated modules. (i) The Primordial Somatic Synchronization Window (FSSP/PSW), whose six layers (S0–S5) set the initial permeability P₀ at birth as a direct function of maternal signature (stress, sonotypic coherence, HRV). (ii) The Q Archive, a subcortical relational index built via absorbed oxytocin, with terminal lucidity as empirical anchor. (iii) Epigenetic modulation OXTR_available(ε) = e^(−κ·ε), orthogonally extended in v5 with the Syt4 secretion-brake gate. (iv) A Mediator ↔️ Primary Protector switching threshold at θ ≈ 170 with hysteresis. (v) Clinical extensions to relational depression, dissociation, autism spectrum disorders (as prenatal miscalibration), and Alzheimer's disease (as late Q collapse via glymphatic-clearance failure).Validation. Six simulation scenarios; six of seven pre-registered predictions numerically confirmed. Bifurcation demonstrated at θ ≈ 170 with inflection shift under ε(m). Sobol sensitivity indices support the composite Collapse Parameter weights (Pc, Eq. 13).Conclusions and next steps. Qualia, so formalized, is quantifiable, simulable, and falsifiable. The paradigmatic reorientation the model implies is not "raise the oxytocin signal" but "optimize the acknowledgement" — a single high-intensity ACK closing the circuit is more valuable for long-term sustainability than many weak signals leaving the OXTR receptor methylated. Testing proceeds through a tripartite C_neuro measurement protocol (GSR + subjective conflict report + Heartbeat-Evoked Potential) under induced-conflict paradigms, stratified by ε(m), P₀, and epigenetic age acceleration.Keywords. qualia · dual neuroception · Katharós Vector · t_KR · epigenetic modulation · dual-attractor · Elena Constant · FSSP/PSW · latch coefficient · retrograde ACK · terminal lucidity · Alzheimer's disease.