Homeostatic Aging Theory: An Integrative Model of Dynamic Degradation, Convergent Feedback Loops, and Social Traps

Contemporary gerontology faces a paradox: the accumulation of molecular-cellular data on ageing is not accompanied by a unified predictive model capable of linking evolutionary biology, metabolism, and socio-behavioural factors. This paper presents the Homeostatic Aging Theory, which postulates that ageing represents not linear wear-and-tear but a systemic bifurcation wherein the dominant role of negative feedback loops (restoration) is supplanted by positive feedback loops (degradation). In contrast to previous formulations that positioned ischaemia as a universal executive mechanism, the present research reconceptualises it as a critical convergent node and tissue-level amplifier, interacting with parallel, hypoperfusion-independent pathways of degradation (proteostatic collapse, epigenetic drift, direct glucotoxicity, nutrient-excess-driven mitochondrial oxidative stress). The theory introduces the concept of a "convergent homeostatic node" and demonstrates how socio-ageing traps (chronobiological disruption, hypokinetic stasis, existential vacuum, evening carbohydrate loading) initiate cascades of molecular damage through the prism of maladaptive remodelling, metabolic inflammation, and loss of exo-informational density. By integrating the concepts of the Exo-Informational Phenotype, the circadian antioxidant conveyor, and noospheric archetypes, the model proposes a formalisable matrix of feedback loops that explains the heterogeneity of ageing phenotypes. The conclusion substantiates the theory's potential for shifting maximum human lifespan beyond 120 years and extending healthspan by more than 50 years through multi-level management of homeostatic control loops. Keywords: homeostatic aging, feedback loops, convergent homeostatic node, ischemic-spasmodic cycle, social traps, exo-informational phenotype, archetypal regulation, proteostasis, epigenetic drift, longevity.